Sidra Nazir

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All individuals on drug therapy should receive an evaluation to check for any concurrent medications that may influence the activity of another drug based on their effects on CYP activity. Based on the characteristics of the drug's profile, CYP enzymes can be either induced or inhibited-marking a significant area of concern in patients due to issues with metabolism and clearance. This can cause considerable adverse effects for patients as medications may either accumulate to toxic levels or clear from the system too rapidly, leading to treatment failure. Also, clinicians must be aware of naturally occurring compounds that can alter the actions of CYP enzymes, such as grapefruit juice, nicotine-containing products, and St. John’s Wort, to name a few. Special precautions are necessary for patients who have decreased baseline activity or injury to CYP enzymes.

Additionally, genes play a role in an individual’s natural composition of CYP enzymes and determine their rate of metabolism. Looking into the future of medical advancements regarding pharmacogenomics, genetic polymorphisms of CYP alleles could take precedent in determining the proper therapy and drug dose for individual patients. With this information, we would better be able to predict drug response in patients.

Common cytochrome p450 inducers, inhibitors, and substrates of the primary isozymes mentioned in this article are listed below.

CYP1A2:

CYP2C9:

CYP2C19:

CYP2D6:

CYP2E1:

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